Toxin
Boric Acid Poisoning
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Boric Acid Poisoning
, Boric Acid, Othorboric Acid, Boric Acid Toxicity, Borate, Borax
Background
Boric Acid is a boron compound
Available as a a fine white powder and in up to 5% solutions
Boric Acid and related Borates have been used in various commerical products
Insecticide
(cockroaches, ants)
Topical disinfectant
Mouthwash and toothpaste
Antimicrobial in
Vulvovaginal Candidiasis
Food preservative
Borates are also used in industrial products
Glazes and enamels
Glass fibers
Mechanism
Toxicity
Poisoning
is typically by ingestion, but may also be absorbed via abraded skin
Mitochondrial injury by boron hydroxyl complexes
Pharmacokinetics
Ingestion <100 mg is typically asymptomatic
Severe symptoms at >50 mg/kg
Lethal dose: 15-20 g (2-3 g for infant)
Renal Excretion
Half-Life
: 10-20 hours
Volume of Distribution: 0.17 to 0.5 L/kg
Findings
Poisoning
Gene
ral
Hyperthermia
Cardiovascular Effects
Hypotension
Neurologic Effects
Headache
Lethargy to
Coma
Lightheadedness
Irritabilty
Seizure
Gastrointestinal Effects
Nausea
Vomiting
(blue-green appearance)
Diarrhea
(blue-green appearance)
Abdominal Pain
Mucositis
Dermatologic Effects
Diffuse skin erythema over face, axilla, inguinal region (red-orange coloration similar to boiled lobster)
Rash progresses to
Desquamation
Renal Effects
Acute Kidney Injury
with
Anuria
(due to
Acute Tubular Necrosis
)
Labs
Obtain full toxicologic workup
See
Unknown Ingestion
Comprehensive Metabolic Panel
Liver Function Test
abnormalities (
Transaminitis
)
Metabolic Acidosis with Anion Gap
Osmolar Gap
Boric Acid Levels
Measured in urine and whole blood
Borate levels poorly correlate with toxicity levels
Management
See
Unknown Ingestion
Decontamination
Remove contaminated clothing and wash skin with soap and water (topical exposure)
Consider small bore nasogastric lavage (if recent large ingestion within last hour)
Poor absorption by
Activated Charcoal
ABC Management
Intravenous Fluid
Promotes more rapid excretion
Vasopressor
s (e.g.
Norepinephrine
) indicated in
Hypotension
refractory to
Intravenous Fluid
s
Hemodialysis Indications
Severe Toxicity
Acute Kidney Injury
with
Anuria
Upper Endoscopy Indications
Large ingestion with persistent gastrointestinal symptoms
Medical admission indications
Symptomatic patients (more than mild gastrointestinal symptoms)
Neurologic findings
Acute Kidney Injury
References
Carroll and Yakey (2025) Crit Dec Emerg Med 39(6): 46
Tomaszewski (2021) Crit Dec Emerg Med 35(2): 28
Leikin (1995)
Poisoning
and Toxicology, Lexicomp, p. 870-1
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